Retinopathy Guide: Causes, Symptoms and Treatment Options

What is Central Serous Chorioretinopathy?

central serous retinopathy caused by corticosteroids

By using this site, you agree to the Terms of Use and Privacy Policy. Hypertensive retinopathy — A physician examines the eye with an ophthalmoscope. Infants should be examined again at 8 weeks of age. Certain sympathomimetic drugs have also been associated with causing the disease.

Central Serous Chorioretinopathy

Patients' response to inhaled corticosteroids has some basis in genetic variations. The retina contains many blood vessels. European journal of ophthalmology. People with very high blood pressure and swelling of the optic nerve require emergency treatment in a hospital. Drug, healthcare and patient safety.

Sometimes a fluorescein angiogram is used to confirm the diagnosis. Most cases clear up without treatment in 1 or 2 months.

Patients with more severe leakage and more severe visual loss or longer persistence of disease may be helped by laser treatment to seal the leak. Patients who are using steroid drugs for example, to treat autoimmune diseases should discontinue their use if medically feasible. Any change in steroid drug use in these conditions MUST be under the supervision of a physician. A small number of patients will have complications of laser treatment laser photocoagulation or hot laser which impair central vision, which is why most patients will be allowed to recover without treatment.

Cold laser transpupillary thermotherapy is said to be a lower risk alternative to hot laser, and photodynamic therapy has also shown some promise.

In a small study researchers found that treatment of CSR patients with melatonin benefited both vision acuity and the density of the macular pigment.

Researchers found that although blood levels of lutein improved in CSR patients treated with 20mg daily, macular pigment density did not - however, the condition remained stable while in the control patients given placebo, macular pigment density continued to thin. Our vision wellness recommendations. However, there is very little information in the literature regarding the relationship between topical respiratory corticosteroids and this disease.

The abstracts of both of these studies are also copied for you below. Based upon these studies, one cannot definitively state that inhaled corticosteroids can produce central serous chorioretinopathy, but it is also not possible to exclude such a relationship.

Thus, I am afraid that your question cannot be answered definitively. Because of a lack of data, we cannot make a definitive suggestion to you. What I have done in instances, such as the one you are facing with your patient, is contact the ophthalmologist who is caring for the patient and discuss the issue.

The most obvious reason which you mentioned for this is that the lack of controller therapy would place the patient at risk for greater use of systemic corticosteroids, which are known to exacerbate the illness. Thank you again for your inquiry and we hope this response is helpful to you. The purpose of this study is to investigate the relationship between corticosteroid use and central serous chorioretinopathy CSC.

A prospective, case-controlled study. A consecutive series of patients with acute manifestations of CSC and a control group matched for age, race, and gender were recruited between January and July A detailed clinical history was taken, and fundus examination with slit-lamp biomicroscopy was performed on all patients.

Diabetic retinopathy — An ophthalmologist examines the retina and inside of the eye. He or she uses a lighted instrument called an ophthalmoscope. A dye may be used to reveal leaky blood vessels. Hypertensive retinopathy — A physician examines the eye with an ophthalmoscope. He or she looks for pale or white areas of the retina. These areas are pale because they are not getting enough blood. The doctor also may see bleeding from ruptured blood vessels or swelling of the retina or optic nerve.

Central serous retinopathy — A doctor or ophthalmologist uses an ophthalmoscope to detect fluid between layers of the retina. This fluid can resemble bubbles. Retinopathy of prematurity — In most affected babies, this condition gets better on its own. A small number of more serious cases get worse without treatment. Babies who need treatment are treated in the first few months of life.

It is usually possible to know within months whether there is any significant long-term damage to vision. Diabetic retinopathy — Controlling blood sugar and blood pressure can slow or halt the progress of the disease. Treatments can repair existing damage. Hypertensive retinopathy — Lowering blood pressure often can stop ongoing damage to the retina. However, some existing damage can persist.

Central serous retinopathy — Most cases go away without any treatment within three to four months. In cases that persist, laser treatment is often used. Full vision can return within six months. Retinopathy of prematurity — The first line of defense is regular prenatal care. This helps to prevent premature birth. Premature and low-birth-weight infants should be screened for retinopathy of prematurity if they:. ROP can be caused by or get worse from inadequate or too much oxygen after birth.

Therefore, oxygen levels are closely monitored and adjusted. Diabetic retinopathy — Controlling blood sugar and blood pressure are essential to prevent diabetic retinopathy.

Annual eye exams are crucial for people with diabetes. If you have diabetic retinopathy, you should have more frequent eye exams. Treatment can start before sight is affected, which helps prevent vision loss.

Central serous retinopathy — The possible causes of this disease are not understood. Therefore, prevention is difficult. Central serous retinopathy has been associated with prescription corticosteroid treatment. If possible, limit the amount of corticosteroids you take. Retinopathy of prematurity — No treatment is recommended during the early stages. However, close monitoring is essential. An ophthalmologist should examine high-risk infants before they are discharged from the hospital.

Infants should be examined again at 8 weeks of age. If the disease is active, the infant should be examined every 1 to 2 weeks until he or she is 14 weeks old.

After that, they should be examined every 1 to 2 months. More advanced disease may require treatment. A procedure called cryotherapy uses cold to destroy abnormal blood vessels.

Iamges: central serous retinopathy caused by corticosteroids

central serous retinopathy caused by corticosteroids

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central serous retinopathy caused by corticosteroids

Indocyanine green angiography can be used to assess the health of the retina in the affected area which can be useful in making a treatment decision. In many cases, the fluid collecting behind your retina will go away without any treatment.

central serous retinopathy caused by corticosteroids

Gass JD, Little H. A procedure called cryotherapy uses cold to cirticosteroids abnormal blood vessels. Paralytic strabismus Ophthalmoparesis Chronic progressive external ophthalmoplegia Kearns—Sayre syndrome. Cold laser transpupillary thermotherapy is said to be a lower risk alternative to hot laser, and photodynamic therapy has also shown some promise. Retinopathy means that disease has damaged the retina.